Iga nephropathy occult blood is how?

iga nephropathy occult blood abatement is to allow many patients troubled by things, so first we want to clear the iga nephropathy cause.
iga nephropathy is one kind of mucosal immune function deficiency disease, IgA nephritis mainly by iga antibodies or immune complex deposition in the glomerular mesangial area, complement components, leading to the inflammatory reaction, activation of the complement system , which led to the obstacles of coagulation and fibrinolysis, resulting in a series of inflammatory mediators, and promote kidney tissue ischemia, hypoxia and damage of capillary endothelial cells to attract the blood circulation of inflammatory cell infiltration, and release of inflammatory mediators of the pathogenic glomerular basement membrane damage, kidney increased permeability, decreased ability to block the red blood cell leakage, clinical hematuria, kidney tissue is impaired.
The elimination of the occult blood in here to remind the appearance of occult blood only one kidney damage, commonly used hormones to control the clinical, hormonal control occult blood by inhibiting the immune response, but can not solve the root causes of kidney damage, can not be cleared immune complexes, immune complexes, there has been constant will cause kidney damage, the patient's own immune disorders lead to hypersensitivity, resulting in the aggravation of the disease. Therefore, even if clinical symptoms disappeared, the late event of the predisposing factors will relapse, most patients are distressed, the condition repeatedly delayed healing will only result in further deterioration of the disease.
Therefore, for the treatment of the disease on the one hand, control of occult blood some of the clinical presentation, the most important thing is to clear the immune complexes deposited in the kidneys, at the same time also pay attention to the low-salt diet, low protein diet, soy products, seafood, beef forbidden to eat the lamb, spicy foods, the diet with the treatment, the same can get a good effect, so as to achieve the fundamental purpose of the treatment.

Causes of female uric 10 years ago what is 2 plus?

Hematuria can be divided into early end of hematuria, hematuria, total hematuria. Detailed methods are three cups of urine testing. Take 3 a clean of glass cup, patients each urination Shi, is divided into Qian, and in the, and Hou 3 paragraph urination, respectively row into 3 a glass cup in the, if 1th Cup in the for hematuria, remaining 2 cup normal, is for early hematuria, reminds urine liquid in the blood from urethral; if 1th, and 2 cup in the no hematuria, only 3rd Cup has hematuria, called end is not hematuria, reminds lesions in Hou urethral, and prostate, and bladder neck and triangle; as three Cup in the are has hematuria, called full hematuria, reminds lesions in kidney, and Ureteral, or for bladder within pervades bleeding.
Causes of hematuria, female causes of hematuria in 10 years what is 2 plus? Mainly has following several aspects: ① urinary system diseases such as nephritis, stones, infection, tuberculosis, cancer, deformities, injuries, etc. ② urinary tract organs in the vicinity of lesion, such as prostatitis, prostatic hypertrophy, pelvic inflammatory disease, appendicitis, etc. ③ systemic diseases, such as infectious diseases, blood diseases, cardiovascular diseases, and so on. ④ drug-induced factors, such as taking sulfa drugs, anticoagulants, or injection of mannitol. ⑤ hematuria after exercise.
Hematuria associated with other systemic symptoms, you can thus analyse the causes of differences between serum uric. Current adoption of urinary erythrocyte morphology of the public prosecutor's Office, to assume that sources of hematuria, in line with the rate of 90% per cent. Detailed approaches are centrifugal urine, placed on a slide, use a microscope to observe the morphology of urinary red blood cells in. Source of Glomerular hematuria, red blood cell deformation of red blood cells in the urine, that red blood cells emerged a variety of forms, such as a ring-like, strawberries, water droplets, even broken red blood cells, reminding hematuria due to Glomerular lesions as a result. Female hematuria caused by what is? non-source of Glomerular hematuria, red blood cells in the urine is normal or maybe normal form.
In addition, the identification of gross hematuria should be matched to hemoglobinuria. Hemoglobin in urine was due to haemolytic disease, plasma free hemoglobin in increased renal threshold was crossed, from glomerular filtration, who entered in the urine. Hemoglobin in urine color can be red, brown or even black color. Identification of hematuria and rely mainly on the microscope the public prosecutor's Office, hematuria urine contains large amounts of red blood cells, hemoglobin in the urine without red blood cells, this is the fundamental difference between the two.

Iga nephropathy protein, creatinine 300!

Treatment for six months no negative IgA nephropathy is the glomerular mesangial iga iga deposition-based, with or without other immunoglobulins in the glomerular mesangial deposition of primary glomerular disease . The clinical manifestations: recurrent gross hematuria or microscopic hematuria may be associated with varying degrees of proteinuria in some patients with severe hypertension or renal insufficiency.
You are in the stage of renal insufficiency, abnormal indicators of increased renal pathological damage caused by kidney filtration and detoxification function decline. Now is how treatment? Treatment not only emphasis on the manipulation of the clinical indicators and, more importantly, from the start of the etiology of the disease, able to effectively repair and reverse the pathological damage of the kidney. Such a condition to get the fundamental manipulation, now active treatment effect is good, this is a chronic progressive disease, we recommend aggressive treatment as soon as possible, in order to avoid the disease is continuous progress to miss the timing of treatment.

What is the latest drug treatment of iga nephropathy?

Anticoagulant: the increase of inflammatory cells in the blood, to a certain extent, increase blood viscosity, which increases the micro-thrombosis in the glomerular capillary. The use of anticoagulants to prevent the formation of microthrombi, blood flow, further lay the foundation for the repair of mesangial cells.
What is it? Latest treatment iga nephropathy drugs and vasodilators: Why do you want to vasodilators it? Iga immune complexes after stimulation cause kidney capillaries, mesangial cells, ischemia and hypoxia the vasodilators's purpose is to improve renal blood circulation, relieve kidney ischemia and hypoxia of the intrinsic cell repair for the next phase in mesangial cells provide a good environment.
What is the treatment of iga nephropathy medicine? Degradation: the increase of inflammatory cells in the glomerular capillaries and blood vessels in the micro-thrombosis and the iga immune complex deposition will lead to glomerular capillaries, mesangial cells of the sediments aggregation and extracellular matrix increase, So we must use drugs to degradation of the deposition of extracellular matrix excreted with the urine to avoid glomerulosclerosis.

Earlier you can cure IgA nephropathy?

Attention problems in diagnosis and treatment of IgA nephropathy in process
I should be regular follow-up of IgA nephropathy urine protein quantitative, blood pressure, urine, renal function and renal b-.
II reducing proteinuria, should control the protein in urine in patients with IgA nephropathy to below 0.3 g/24 hours
In patients with recurrent attacks of III eye of IgA nephropathy with hematuria, tonsillectomy;
V positive control of hypertension, blood pressure should be reduced in patients with hypertension 130/80mmHg or 125/75mmHg the following;
Diagnosis of primary IgA nephropathy in patients with IgA nephropathy daily needs to pay attention to? Answers to the above we have, here is the check of IgA nephropathy, on this issue, we work together to get an idea.
Can you cure IgA nephropathy in early stage?
Found in physical examination: the majority of no abnormal signs, and some patients may have a dual-percussion pain in kidney area, swelling and mild to moderate increase in blood pressure.
Accessory examination: mostly mild to moderate proteinuria (<3G/d), hematuria is polymorphic, diversity or mixed. Some patients with elevated serum IGA, serum IGA fibronectin in particular polymers (iga-FN) increased more meaningful. May have reduced creatinine clearance, hematuria effect of nitrogen and creatinine increased. According to the clinical preliminary diagnosis of IgA nephropathy (inflammation), diagnosis of immune pathology of renal tissue must be done.
Medical history and symptoms: upper respiratory tract infections (acute gastroenteritis, peritonitis, or osteomyelitis) after 1-3 days appear vulnerable to recurrent episodes of hematuria by naked eyes, under lasting several hours to a few days later to hematuria, may be accompanied by abdominal pain, low back pain, muscle pain or heat. Some patients found during a physical examination of urinary anomalies, asymptomatic proteinuria and (or) under the microscopic hematuria, a small number of patients have persistent hematuria and proteinuria to varying degrees to the naked eye, and may be accompanied by edema and hypertension.

Creatinine high smoking?

Many smokers know smoking is bad for health, but was unable to give away, then for creatinine high smoking? is the habit of smoking patients it is necessary to "endure pains silently to give away what one loves" out?
According to understanding muscle anhydride high of patients on smoke wine are is to limit of, because smoke wine of poisoned main is on kidney, and vascular of poisoned, smoking, and drinking more more on kidney vascular injury on more large, more early of increased has renal arteriosclerosis, more promoting renal ball of hardening, so, Shijiazhuang nephropathy hospital nephropathy expert Yang Dongping Director pointed out that, regardless of is renal features normal also is renal features exception muscle anhydride high of patients are to strictly quit ring wine, avoid caused kidney more large of damage.
Thus, for smoking in patients with serum creatinine high does this issue clearly has very clear, except in patients with serum creatinine level of restrictions on tobacco and alcohol to, in addition to serum creatinine high in dietary restrictions:
① vitamin intake to moderate: creatinine high in renal failure patients to choose foods rich in vitamin a, vitamin B2, vitamin c foods. If only people with kidney failure in patients with renal insufficiency creatinine high swollen but no less urine, no patient could not help salt and water, maintain a low salt diet. If you have severe edema, as well as high blood pressure and even heart failure should be strictly limited ban salt.
② to limit the salt: in general compensatory phase of renal insufficiency, with function of nitrogen in patients with serum creatinine is high, and minor clinical symptoms start, but because of injury in the pathology, Visual no high blood pressure, and edema, control the amount of salt intake, respectively, giving low-salt or salt-free diet.
③ protein supply must be reasonable: in order to prevent the continued deterioration of the renal function in patients with creatinine high, should control the protein intake, selection of high quality protein diet rich in high quality.

Causese of Diabetic nephropathy

Diabetic nephropathy is caused by different causes and onset mechanism of insulin in the absolute and the relative lack of that sugar, protein and fat metabolism disorders, chronic high blood sugar, the main clinical manifestations of systemic disease. Can diabetes damage the kidneys of those damages by different means involving the kidney structure, but only glomerular sclerosis and diabetes, also known as diabetic nephropathy, is one of the microvascular complications of diabetes systemic. Diabetic patients in the event of kidney damage persistent urinary protein, the disease is irreversible tend to develop to the end-stage renal failure. Diabetic nephropathy has become the leading cause of death in diabetic patients.
Diabetic nephropathy is more common in patients with diabetes duration of more than 10 years of proteinuria is the earliest manifestations of diabetic nephropathy, the onset mechanism is very complex, yet completely clarified. The research data show that the onset mechanism of diabetic nephropathy is multifactorial, mainly in the following areas:
1, the renal hemodynamic abnormalities
Play a key role in diabetic nephropathy, and may even be the initiating factor.
(1) hyperglycemia, glomerular hyperperfusion, hyperfiltration state, across the capillary wall pressure, so that the expansion of mesangial cells, epithelial cell foot process fusion and produce dense droplets, glomerular epithelial cells from off on the basement membrane. (2) the glomerular basement membrane collagen type Ⅳ messenger sugar nucleic acid increased basement membrane thickening, the final form of diffuse mesangial nodular lesions, the occurrence of glomerular sclerosis.
(3) In the case of pressure, increased protein filtration can also be deposited in the mesangium and glomerular basement membrane, promoting stromal proliferation constitute a vicious cycle, and can cause nodular and diffuse glomerular sclerosis.
2, hyperglycemia
Diabetic nephropathy is closely related to high blood sugar, poor blood sugar control can accelerate the occurrence and development of diabetic nephropathy good glucose control clearly slow down the development. Hyperglycemia and advanced glycation end products increased production caused by the proliferation of mesangial cells increase extracellular matrix, mesangial expansion, glomerular basement membrane thickening.
3, genetic factors
Most diabetic patients eventually kidney disease does not occur, the same, there may be some good long-term glycemic control in patients with diabetic nephropathy. Glucose transporter protein -1 (GLUT1) is the major glucose transporter in mesangial cells. Recent studies have found that diabetic patients from different individuals mesangial cell GLUT1 menu of the differences in the regulation may be one of the factors of some patients susceptible to kidney damage. And diabetic nephropathy also showed family aggregation in some family history of hypertension in diabetic patients, the incidence of diabetic nephropathy is also significantly higher in patients without hypertension, family history. In addition, the incidence of diabetic nephropathy between the different races there are also differences. That show the occurrence of diabetic nephropathy and genetic factors.
4, high blood pressure
With diabetic nephropathy are not directly related to microalbuminuria urine protein of the blood pressure can accelerate the progress of diabetic nephropathy and deterioration of renal function, increased urinary albumin excretion of the original high blood pressure or duration.

How to treat Leg edema with diabetic kidney disease

Patients with diabetic nephropathy are common symptoms of localized edema, which is due to impaired renal system, resulting in the body of water and toxins can not be excluded from the body, caused by the retention. Of edema and proteinuria plus sign is necessary and timely treatment, timely treatment will result in the lesions of various organs of the body functions, resulting in edema and proteinuria is necessary and timely medical treatment.
Diabetic nephropathy is the original or secondary cause of along with the immune regulatory dysfunction caused by elevated blood glucose, a series of pathological changes of intrinsic glomerular cells, a large number of immune complexes in the pathogenesis of deposition in the kidneys leading to glomerular basement membrane thickening, luminal narrowing, blood vessels, microcirculation, leading to renal ischemia and hypoxia, the intrinsic cells of the kidney damage can not stop the protein leak. In fact, the abnormal protein and the amount of urine is not only a disease indicators, but as an independent factor involved in the process of kidney disease. The study also found that the level of a variety of nephrotic proteinuria and chronic renal failure is closely related to the rate of progress, enables the protein to reduce the therapeutic interventions are beneficial to slow down the progression of kidney disease.
Protein loss of the formation of excessive hypoproteinemia, the loss of a protein from the glomerular capillaries, resulting in plasma colloid osmotic pressure (plasma colloid is a plasma protein), and the important role of the plasma colloid is to regulate the blood vessels inside and outside the water exchange, it appears that the osmotic pressure will lead to water retention in the interstitial to stay, cause edema. The same time, hypoproteinemia can cause the effective hypovolemia secondary aldosterone and antidiuretic hormone secretion, increased tubular reabsorption of water, sodium increases, causing swelling.
Symptomatic treatment for the treatment of diabetic nephropathy edema following methods: 1, in patients with edema with diuretics, spironolactone and thiazide drugs available is used in conjunction. 2, appears in patients with heart failure, edema can be applied to digitalis therapy. Sodium 3 patients with severe refractory edema can be used diuretic furosemide. 4, severe hypoproteinemia and edema in patients with intravenous albumin and essential amino acids. 5 patients with severe anemia, edema may be a small amount of blood transfusion.
But in the end, in order to radically improved edema and diabetic nephropathy caused by other complications, the most important treatment is to step up to the kidneys. To confirm the diagnosis of the type of kidney immune and inflammatory response and immune complex deposition in the kidney site targeted drug therapy. To timely protection of residual renal function treatment, we can achieve the purpose of disease control.

Why is edema

Edema is just a manifestation of the symptoms caused by edema, a variety of reasons. Stay up late such as edema, is only a temporary edema, and get adequate rest will return; such as pregnancy edema, slight edema is a normal phenomenon, and only need conditioning can weekdays; such as kidney disease caused by edema, but it can not be ignored, the need for timely diagnosis and treatment.
In the normal human body, intravascular fluid continuously from the capillaries, arterioles side filter out the tissue fluid to the interstitial space. The other hand, the tissue fluid from capillary venules side back absorbed into the blood tube, the two often maintain homeostasis, and thus excessive fluid accumulation in tissue spaces.
So, the main factors to maintain that balance, what does?
(1) capillary hydrostatic pressure;
(2) plasma colloid osmotic pressure;
(3) the mechanical pressure of the interstitial space (tissue pressure);
(4) tissue fluid colloid osmotic pressure.
If the factors that maintain fluid balance disorder, may give rise to edema.
So, of edema factor and what it?
(1) the presence of sodium and water: If the secondary aldosteronism psychosis;
(2) capillary filtration pressure is increased: at right heart failure;
(3) increased capillary permeability: such as acute nephritis;
(4) plasma colloid osmotic pressure: usually secondary to reduce serum protein, such as chronic nephritis, nephrotic syndrome.
Thus, to maintain fluid balance is the key to the prevention of edema. Once the imbalance will cause varying degrees of edema. Some edema factor is controllable, proper rest and conditioning can be, and some edema factor is not controllable, need to go to hospital for a detailed examination, if the disease but also to early treatment.

Life Expectancy of stage 3 diabetic nephropathy

A long-term effective control of diabetes: high blood sugar is the basic factors for the development of diabetic nephropathy, early detection of diabetes and proper treatment of the as much as possible so that the three substances metabolism back to normal is critical. Control the blood sugar at normal levels, often make the early pathological changes to restore, but of such progress to clinical renal play, even if the strict control of blood glucose, the effect is also poor. Close to uremia of diabetic nephropathy can occur the following phenomena: (1) Renal sugar group clearly increased, it is not urine to determine the degree of glycemic control, the surface should be subject to the determination of blood garnet, increased the difficulty of the conditioning dose of insulin and hypoglycemic agents ; ② certain metabolites in uremic fatigue reducing, caused by the use of copper sulfate reduction assay urine false positive; ③ The uremic patients loss of appetite, eating less, even take the kidney itself off can decline to insulin, reduce insulin requirements, and prone to hypoglycemia should be readily conditioning dose; ④ generally not suitable for re-use of oral hypoglycemic agents, the surface should be used insulin. Oral hypoglycemic agents can be used with caution worse diabetes non-insulin-dependent diabetes. Biguanide easy to induce lactic acidosis, should not be used; tolbutamide (D860) and sugar Fit is safer, but there are reports of hypoglycemia, the application still need to be closely observed.
Two, the positive treatment of hypertension: antihypertensive treatment is very important for the slow rate of decline of glomerular filtration rate. Blood pressure control, proteinuria discharge also decreased. Make the blood pressure dropped to 16.8/l1.5 kPa (126/80mm Hg), and in that time to treat high blood pressure is more important than the treatment of high blood sugar, but both at the same time. Have favored nephropathy selected angiotensin converting enzyme inhibitors, the preparation turned into angiotensin II by inhibiting angiotensin Ⅰ, so that sodium retention to reduce the blood vessels to dilate peripheral resistance decreased, so that the drop in blood pressure. In addition, because the glomerular human ball, efferent artery dilatation, glomerular improve the blood supply, thereby reducing the glomerular progressive damage and improved renal function, commonly used drugs Capoten, said Ning sneeze can also be used nifedipine, the group of Nepalese peace, compound antihypertensive tablets and other treatment.
Third, adjust your diet: reducing protein intake, not only for renal insufficiency favorable, but also help to reduce the emission of proteinuria. The daily protein intake does not exceed 30 to 40 grams. Selection of high quality protein, such as milk, eggs, meat. Soy products and so should be limited.
Dialysis and transplantation: garnet urine disease of kidney disease uremia should be carried out peritoneal or hemodialysis treatment. Kidney or pancreas - kidney transplantation for the treatment of diabetic kidney late the most effective way. After transplantation, allows glycosylation of hemoglobin and serum creatinine, phenol levels return to normal.
Fifth, do not use harmful drugs: the kidneys, such as gentamicin, streptomycin, amikacin, neomycin, etc..

Anemia and renal insufficiency have a relationship?

Anemia, it seems that with renal insufficiency nothing, but in fact, this really true? Most people believe that, so-called anemia, nutritional anemia caused by malnutrition, as long as maintenance, adding nutritional supplements, it should be will be improved. Some doctors only folic acid, iron and blood oral solution for treatment. So, anemia and renal dysfunction how kind of relationship, let's take a look.
In many cases, the primary disease symptoms can be very subtle, mild, as usual cold, complicated by mild or occult glomerulonephritis may be due to repeated colds, but the symptoms are light and did not care. After a long process, the progression to chronic renal insufficiency, and the emergence of the digestive tract as the main symptoms of renal anemia.
Kidney chronic nephritis or acute lesions of chronic renal insufficiency, erythropoietin hormone level will drop, the ability of the patient's body to manufacture red blood cells decreases, so the patient will show symptoms of anemia. Combined with patients with no apparent history of kidney, part of the patients are often mistaken for iron deficiency anemia, refractory anemia, aplastic anemia, which delays the diagnosis and treatment of disease.
Renal anemia is due to various causes kidney damage, renal insufficiency, and reduced renal function, excretion of reduced metabolites, retention increased. These chemicals inhibit erythropoietin generated on the one hand, on the other hand, inhibit the activity of erythropoietin, resulting in the destruction of the red cell membrane and shorten its life. Due to renal insufficiency and long-term anorexia, diarrhea, caused by lack of iron, folic acid, vitamin B12, and ultimately lead to renal anemia.
Through the above description, I believe that the anemia have sufficient knowledge, you can see, anemia is also common symptoms of kidney disease, even in normal urine, have to do routine urine examination. Once the urine proteinuria or hematuria, associated with abnormal blood pressure, then tend to have the possibility of renal anemia, and must continue to do blood, kidney function, kidney B ultrasonic examination, so that we can quickly diagnosed. Therefore, anemia appears to be simple, but they can not be overlooked.

What are causes of renal dysfunction?

What are causes of  renal dysfunction?This is the concern of many patients with renal insufficiency, hospital experts your questions are described in detail the main reason for renal dysfunction.
The main reason for renal insufficiency, including two major aspects: the disease lead to renal dysfunction and drug induced renal dysfunction.
(1) the disease lead to renal failure
Renal insufficiency is the development of kidney disease to late renal partial or complete loss of the pathological state. A variety of chronic kidney disease can lead to renal function decline, and the eventual development of renal insufficiency, renal insufficiency reasons. May lead to chronic renal insufficiency, some chronic diseases, including primary glomerulonephritis, diabetic nephropathy, hypertension, kidney disease, kidney the quality tubule kidney disease, polycystic kidney disease and other illnesses.
(2) drug induced renal dysfunction
Found in a survey of the number of patients with renal insufficiency, inappropriate use of nephrotoxic drugs long-term damage to the kidneys, accumulated up to a certain degree of renal insufficiency. That is an important factor to cause renal dysfunction causes, such as painkillers, analgesic film containing non This cefoxitin taking long-term continue to damage the kidneys, causing chronic interstitial nephritis, and then the development of renal insufficiency; In addition to outside the inappropriate use of gentamicin and other drugs to cause the key to the causes of renal insufficiency.

Renal insufficiency, diet needs attention?

This is particularly concerned that many patients with renal insufficiency and their families, the following experts for everyone to do the details, hoping to give everyone's life.
Renal insufficiency, diet needs attention? Had renal insufficiency to eat what?
One kidney patients how to restrict salt intake regardless of what kind of kidney disease, as long as there is edema, it is necessary to restrict salt intake. Edema, a day should be limited to less than 1 g in serious cases, salt-free diet. Until urine protein to reduce edema, the amount of salt may be appropriate to increase, but should also be a day over 5 grams. The purpose of the low-salt diet to reduce body of water, sodium retention, edema and decreased blood pressure. 1 to 2 grams per day of salt (trumpet toothpaste cap filled with about 1 gram), with no dishes, because it put the dishes are basically tasteless, to change the eating. Morning sweets; noon to 1 g of salt on the small dish with vegetable sauce to eat. Thus, the total intake unchanged, but tasted salty, and can stimulate appetite. But we know that low-salt diet, can not eat pickles, pickles, mustard, salt, bread, fritters, do not eat seaweed, rape, spinach, fennel, celery, day lily flowers, radish. Higher because these foods the hectogram in sodium amount. Eat a lot of these foods, it is equal to an increase in the amount of salt
2, kidney patients how to intake of vegetables, fruits, kidney patients, general urine is normal, no oliguria and renal failure, should eat more vegetables, fruits, in order to supply adequate vitamin. Such as patients with decreased urine output, in particular, is less than 500 ml a day, they have selective consumption of vegetables and fruits. As vegetables, fruits, generally potassium rich, while patients with kidney disease oliguria, serum potassium levels significantly increased. Hyperkalemia can cause cardiac arrest, life-threatening. Vegetables, fruits, cereals are foods rich in potassium, high potassium fruits watermelon, banana, pineapple, mango, dates, melons, etc.; amaranth, spinach, celery, carrots, vegetables, high potassium, bamboo shoots, potatoes and so on. Renal patients in the oliguric stage should eat less, if the use of diuretics, potassium low, you can eat, especially fresh fruit juice rich in potassium, you can use oral potassium. Hami melon is rich in potassium, contains about 250 milligrams of potassium per 100 g flesh. High levels of potassium in the body can trigger heart disease, bradycardia, conduction block and other life-threatening. Renal failure, glomerular filtration rate, tubular function decreased and potassium metabolism disorders, but not in a timely manner to the excess of potassium excreted, resulting in hyperkalemia. Therefore, the clinical manifestations of renal failure in patients unfit for human consumption of cantaloupe
, Uremic patients to make arrangements to three meals a day diet (1) the main staple food of the breakfast: the choice of fresh milk 100 ml plus 50 grams of sugar, or lotus root starch plus sugar water prepared and bowl a few blocks, Jiamai starch biscuits, or oil fried potatoes, a small amount, or egg. The discretion to add lotus seeds, dates and so on. Some people like to add some Kaobai Shu, raisins, chicken, burned potatoes and other. (2) the staple food for lunch: wheat starch, bread, cakes, dumplings, steamed, ravioli, etc. can be used. (3) the staple food for dinner: the selection with the same lunch staple food
4, renal failure patients should not eat more of chronic nephritis in which the sea (water) due to loss of protein in urine, and renal function is still good blood non-protein nitrogen concentration is not high, can add a lot of protein. When renal insufficiency, and must be accompanied by decreased urine output, high-protein food unfit for human consumption, otherwise, will happen after eating uremia. Therefore, a higher protein content of the sea (water) products, such as sea cucumbers, dace, Le fish, yellow croaker, tiger fish, mandarin fish, etc. should not eat
5, renal failure patients do not eat meat renal failure patients, protein intake should be limited. Because of its nitrogenous waste in the body can metabolize, and with the urine through the kidneys. Renal failure (oliguria), decreased urine output affect the excretion of waste, the accumulation of these waste-prone uremic symptoms. Such as ham, Tian chicken, chicken, pigeon, quail, bird meat and other meat foods high in protein, are appropriate to eat.
These are the experts attention to the details of matters to do with renal insufficiency diet, I believe you also understand, and usually must be reasonable scientific diet, so be conducive to the treatment of diseases, and promote the rehabilitation process.

Polycystic kidney disease in patients with common complications?

We should note that polycystic kidney disease is a chronic disease, early treatment in addition to the product level, is to control, monitor the development and polycystic kidney disease symptoms appear. Suffering from polycystic kidney disease, many people in the early symptoms is clear that only through the CT scan in order to accurate diagnosis. But asymptomatic, very few people to do CT scan.
The following main points:
An abnormal emotional fluctuations: abnormal fluctuations in emotions and in particular the long-term excessive depression or panic.
2, waist, abdominal discomfort: the waist, abdomen local discomfort and abnormal unexplained high blood pressure or renal function.
The hematuria cycle attack: hematuria cycle of attack or mild urinary protein (with nephritis differentiated).
4, lead to excessive fatigue: overworked, including excessive physical labor, excessive mental work and lack of physical exercise or exercise practices, improper strength, the Housing excessive labor.
Pollution of the environment of life: the people who live in serious pollution of the environment for a long time, or exposure to toxic chemicals (including taking nephrotoxic drugs) or ray.
6, have a family history: the family members of someone suffering from polycystic kidney disease, renal cyst. Polycystic kidney disease is often accompanied by how the capsule of the liver, liver cyst, splenic cysts, pancreatic cysts, cerebral aneurysms, abdominal aortic aneurysm, gastrointestinal diverticula and other diseases, so when their own or family members of these diseases should pay special attention to the polycystic kidney disease, renal cyst.
What are common complications of several aspects of the early symptoms of polycystic kidney disease onset by the above-mentioned patients with polycystic kidney disease, is conducive to early prevention and treatment of the patient. Thus once own the above symptoms need for timely medical treatment. So as not to affect the recovery of the disease the best time. Pay more attention to some abnormalities in their lives, contribute to early detection. An early manifestation of the above aspects of polycystic kidney disease complications, the need to attract everyone's attention.

Why do we suffer from polycystic kidney disease?

Polycystic kidney disease will have a huge impact to the health and life of the patients, many patients with polycystic kidney want is not suffering from polycystic kidney disease like. So why do we suffer from polycystic kidney disease?
Why do we suffer from polycystic kidney disease?
Renal cyst is due to the formation of the cyst epithelial cells of renal tubular epithelial cell variation, and abnormal secretion of cystic fluid, making the formation of cysts and cyst fluid secretion and exclude imbalance. Cyst age increases gradually increasing cyst epithelial cells secrete cyst fluid with no discharge channels, the cyst gradually increasing, cyst rupture will result in acute abdominal infection, while the long-standing oppression of the renal parenchyma will inevitably lead to renal damage, Therefore, treatment for cysts, can not simply go to reduce the cyst, the key is to control the cysts to reduce the cyst on the basis of increased again, to prevent recurrence. Than 3cm cyst can be a positive program of traditional Chinese medicine taken by the infiltration of the kidney area of ​​the skin directly to the cyst, an expansion of the wall of blood vessels, away the cyst fluid, in order to shrink the cyst, is against the wall in a the activity of the epithelial cells, inactivated, it no longer secrete the cyst fluid, and thus control the growth of cysts, for therapeutic purposes.
Adult polycystic kidney disease occurs mainly in adults over the age of 30-50, and adult polycystic kidney disease is more common in clinical practice, the development of renal failure in autosomal dominant polycystic kidney disease late.
Polycystic kidney disease, according to their genetic characteristics, divided into adult polycystic kidney disease and infantile polycystic kidney disease (also known as the two types of autosomal recessive inheritance).
Patients with adult polycystic kidney disease manifests itself in bilateral renal enlargement, cortex, medulla multiple fluid cyst formation and increasing secondary to impaired renal function. Can affect multiple systems, such as the digestive system, cardiovascular system, central nervous system, reproductive system, the formation of liver cysts, splenic cyst, aneurysm, heart valve abnormalities. However, according to the latest research shows that the fetus, including any period can be onset.
Polycystic kidney disease is caused by?
Cyst genetically modified tubular epithelial cells and the wall of cell metabolism, so that:
① cyst gene tubule cells, Na-K-ATP enzyme, so the increase in the amount of fluid within the tubule, and the proliferation of wall cells to secrete liquid;
② small tubular epithelial cells continued proliferation, the formation of polyp-like lesions caused by tubular obstruction. Obstruction in front of the tube, fluid retention, expansion into the cyst. In addition, polyp-like substance formed by the proliferation in the cyst neck can be caused by semi-obstruction, so that the liquid is not easy out, then expand the cyst;
Stimulate cyst gene ③ infection and toxin in the small tube, change the tubule cell metabolism, or even directly cause epithelial cell necrosis, and necrotic cell shedding may also cause obstruction, and necrosis after regeneration also promote cell proliferation.
The ④ tubule epithelial cells and wall cells to produce a series of biologically active substances to promote the secretion of the cyst fluid;
The ⑤ abnormal extracellular matrix, and changes in basement membrane and wall conditions, the development of cyst formation;

Renal cyst is a serious disease?

Renal cysts are common people a kidney disease, it will have a huge impact to the health and life of the patients, renal cysts is a serious disease? Let us look at The experts have the answer.
Renal cyst is a serious disease? Experts give us replied to the degree of renal cysts with many aspects of the situation, but no matter what, as long as we adhere to treatment, renal cysts can recover, just the length of time . Want to know the renal cysts, it is necessary to clear the renal cysts:
Renal cysts are more common in adults over the age of 50, usually no obvious symptoms or symptoms of late. Early neither pain, hematuria, and urine tests are also no exception. Majority in the larger tumor when found. Touch the smooth cystic mass in the abdomen, can be used in strong light irradiation, children or patients with a thin abdominal wall, the cyst can be opaque.
Renal cysts, including the solitary renal cysts, congenital polycystic kidney disease and congenital multiple renal cysts. Patients with renal cysts are usually no obvious symptoms of kidney disease, kidney on the long one or a few cysts found only by B-ultrasound. This is the case of renal cyst, the patient to pay attention to such cases, if not as soon as possible the treatment of renal cysts is likely to deteriorate, resulting in the emergence of uremia.
Had renal cysts also hematuria. Hematuria can be expressed as microscopic hematuria or gross hematuria. The onset of cyclical. Attack of low back pain, often exacerbated by strenuous exercise, trauma, infection can be induced or aggravated. The causes of bleeding because of the wall below the number of arterial pressure to increase co-infection, so that the wall of blood vessel rupture due to excessive traction.
Many patients are more afraid of kidney disease hazards force, life depression, but do not know this life is very easy to give body to the negative treatment.
For small renal cysts, the symptoms do not need to do any treatment, but should be regularly reviewed to observe the cyst continues to increase. Those who are asymptomatic should be regular urine tests, including urine, urine culture, a kidney function tests every six months to one year, including the endogenous creatinine clearance rate. Since infection is an important cause of the deterioration of the disease, urinary tract trauma examination is not very necessary, do not. The role of renal cyst puncture is not only susceptible to infection, easy to relapse, but also by the observation can not delay the occurrence of renal damage.
Renal cysts is a serious disease, everyone should know it, If you are in the renal cysts there any questions in a timely manner online consult our experts, our experts will be explained in detail. Here, I wish the majority of renal cysts in patients with a speedy recovery.

How to treat Simple renal cysts ?

 Most of the small size of this disease, asymptomatic, does not affect renal function, and natural changes slowly or not what happens, it is generally no treatment, but should be reviewed once every six months to a year.
May be considered more than four centimeters in diameter is relatively large renal cysts, puncture fluid, and then inject tetracycline or ethanol line Cyst treatment to prevent relapse. Preoperative need to check the platelet count, bleeding, clotting time, B-type ultrasonic guidance of B-ultrasound probe (intervention B ultrasound) Downlink sclerotherapy of renal cysts.
Will puncture needle into the center of the renal cysts, the first out of the cyst fluid, the best pumping net, and then injected extracts the amount of 1/4 the amount of absolute alcohol, so that patients change position as much as possible, so that the liquid with the wall full access five minutes after taking the liquid, the pin was removed after observation, review after 1 month.

Renal cyst back pain serious

Renal cyst back pain serious? How to treat? Renal cysts is a common clinical renal cystic disease, the disease early with no obvious symptoms, but cysts increases with age, if you do not take the treatment, increasing cystwill affect renal function, followed by a series of clinical symptoms, back pain is one of the common clinical symptoms. Renal cyst back pain serious? How is it treated?
The experts pointed out: the treatment of renal cysts, Western medicine commonly used surgical therapy until the cyst diameter greater than 5cm puncture fluid technique, decortication, large cysts within the cyst fluid aspiration to achieve short-term narrow the purpose of the cyst. The long term, due to the removal of large cysts, the growth of small cysts to free up space, small cysts or rapid growth, once again pose a threat to renal function. Do not recommend surgical treatment of renal cysts. Integrative Medicine can be used in the treatment of renal cysts, and kidney area in vitro penetration of the drug directly transported to the kidneys, high utilization of drugs, which can effectively alleviate the state of renal ischemia and hypoxia, to accelerate renal cyst capsule fluid back to the absorption, and inhibit the secretion of the cyst fluid , and ultimately shrink the cyst does not relapse. At the same time as the case supplemented with Western medicine, to prevent the inflammatory response, play to win a good environment for Chinese medicines.
The majority of patients with chronic kidney disease early symptoms or fewer symptoms; As the disease progresses, the gradual emergence of varying degrees of symptoms. No matter what kind of symptoms, are a group of long-term role of the inflammatory response caused by kidney natural cell damage, most patients did not have timely and effective treatment and lead to protracted illness increased the cure rate is low, recurrent disease. Therefore, renal cysts found that prompt treatment is the key, if there are questions on treatment, you can directly call the hotline, one-on-one communication with experts, tens of thousands of kidney patients in a professional guide treatment!

Polycystic kidney disease treatment how good?

Polycystic kidney disease is a hereditary cystic kidney disease, belongs to the occult disease, early symptoms, but cysts will increase over time, if you do not take timely treatment, may affect renal function,severe cases can cause kidney failure, a serious threat to people's health.Patients and their families as a concern for the treatment of polycystic kidney, polycystic kidney disease how cure?
The treatment of polycystic kidney disease is nothing more than Chinese medicine treatment and Western medicine treatment at home and abroad to take decortication surgery and pumping the liquid curing surgery is the only Western medicine treatment measures for the treatment of polycystic kidney disease, but only an expedient measure, Western medicine treatment to ease the cyst-related symptoms of pain and high blood pressure, mainly the removal of large cysts, reduce the pressure of the cysts on the kidneys, also reduced the fracture, the risk of bleeding, but after all other small cysts around the rapid enlargement vacated growth space, other small cysts due to the surrounding pressure to reduce the development of faster, and never subsided, renal function will continue to aggravate, not solve the fundamental problem.
Shijiazhuang kidney disease hospital in order to overcome this limitation, and create the characteristics of traditional Chinese medicine treatment of polycystic kidney disease topical therapy of micro-based traditional Chinese medicine penetration penetration blocking renal fibrosis therapy, the meridian through the kidney area targeted positioning, targeted therapies for cystic lesions. Pressure decreased by expansion in polycystic kidney blood vessels surrounding the vessel wall, accelerating the wall of blood circulation to promote the increase of wall permeability; to promote within the wall of blood vessels; reversal of the pressure difference due to intravascular pressure higher than the intracapsular pressure; accelerated cyst fluid of water to the blood vessels back to the absorption. Of traditional Chinese medicine at the same time micro-penetration therapy of active substances to inhibit the continued secretion of the epithelial cells of the cyst wall; to prevent the wall of blood vessels and cysts continue enlargement. Infiltration therapy of active substances in micro-based traditional Chinese medicine treatment to improve the status of the vascular wall and the cyst cycle disorders, to soften the wall of the external wall of the permeability and inhibit the role of the wall cells secrete. So as to restore normal kidney function. The treatment in our hospital has been a large number of patients with polycystic kidney embarked on the road to recovery.

Symptoms of Polycystic kidney disease

Polycystic kidney disease in patients with advanced symptoms? Polycystic kidney disease is a hereditary disease, is to grow a lot of large and small cysts in the kidneys of patients, with the increase of cysts lead to kidney structure and function decline, causing great harm to the human body.
Polycystic kidney disease if sooner or later stage is not effective treatment, causing patients to miss the best treatment period, his condition had deteriorated into the uremic phase, resulting in more damage. Timely understanding of polycystic kidney disease with advanced symptoms of what is necessary.
Polycystic kidney disease in patients with advanced symptoms generally include:
, Kidney increased in patients with kidney volume increased approximately 10 times the normal kidney. The morphological abnormalities, such as the renal pelvis shaped, the complete structure of the cone of the renal papilla and renal damage. Sleek, section shows fusiform or columnar cyst, the radial distribution. Epithelial cells were cylindrical, and collecting duct epithelial cells. Renal pelvis and calyceal expansion of the renal parenchyma compression and narrower, smaller. Themselves or others can be touched to the abdominal mass and play the sexy, abdominal distension, sometimes hidden, dull sense of fatigue, tiredness, decreased physical, emotional irritability. Squeeze cysts, adjacent organs, gastrointestinal symptoms, loss of appetite, or abdominal distention after eating, the body is gradually emaciated.
2, hematuria due to kidney cyst volume increases lead to cyst rupture, and thus give rise to the phenomenon of hematuria. The main reason leading to the rupture of renal cysts may be strenuous exercise or overworked, this situation must be timely medical treatment, or the cyst will rupture, the condition will deteriorate.
3, polycystic kidney disease in patients with renal cysts are spherical, and multiple sizes of the cysts, which also contributed to a factor of polycystic kidney disease in patients with renal enlargement.
Final polycystic kidney dirty as the cyst increases renal units completely damaged, have caused the loss of renal insufficiency and uremia, renal.
5, the occurrence of retinal lesions and other complications due to hypertension lead to retinal arteriosclerosis, retinal blood supply to decline; a lot of hematuria led to the Hb sharp decline, further aggravating the original damage on the basis of optic neuropathy and blood supply, leading to the occurrence of secondary optic atrophy. .

Diet of polycystic kidney

The daily care of patients with polycystic kidney need to pay attention to what is it? Polycystic kidney disease is a common hereditary kidney disease, in addition to the proper medical treatment, daily care is also critical. Below, I will introduce what the daily care of patients with polycystic kidney work?
Renal hypertension should be regularly measured blood pressure, bed rest time according to the increase in blood pressure changes.
2 know the patient's diet according to the patient's specific condition, such as patients with renal insufficiency should pay attention to improve the heat, high-quality low-protein diet.
Sooner or later, and postprandial maintaining oral hygiene, bad breath, reduce nausea, prevent bacteria and mold students.
4. Do patients with polycystic kidney, skin care, the prevention of skin infections, bedsores and related complications, an important task, due to the deposition of uremic frost Pifu stimulate the patient often itching and discomfort, and affect sleep, and caught nonintact Cardiff vulnerable to infection.
5 so should wash with warm water and scrub, keep Pifu clean, avoid using soap and alcohol. Frequently changes of clothes, blankets. Patients with severe edema, but also should pay attention to the protection Pifu be frequently changed the decubital, massage of pressure areas and prevention of bedsores.

Polycystic kidney disease the most common hazard is?

Polycystic kidney disease is a common hereditary renal cystic disease. No obvious symptoms of polycystic kidney disease early, When the cyst is gradually increasing, the oppression of the normal kidney tissue and can cause lower back pain, and occasionally patients can touch the abdomen with a soft block with your fingers, are polycystic kidney disease occasionally cause repeated episodes of hematuria, or pyelonephritis. In most cases, polycystic kidney disease has no symptoms, unless the cyst was finally replaced by too much kidney tissue, causing chronic renal failure.
What is it? Polycystic kidney disease is the most common hazards and polycystic kidney disease are usually in for a checkup for some other reason was found. Many patients with polycystic kidney did not know they have the disease. However, severe polycystic kidney disease is extremely rare. In the U.S., about 2% of chronic renal failure disease caused by polycystic kidney disease.
What is it? Polycystic kidney disease is the most common hazards and polycystic kidney disease has become chronic kidney disease accounted for a large proportion of kidney disease, so more and more the attention of the majority of patients. Polycystic kidney disease is divided into autosomal dominant and autosomal invisible genetic, familial aggregation, male and female incidence probability is the same, from generation to generation, never inter-generational. Polycystic kidney renal insufficiency, there will be some complications, such as high blood pressure, nausea, vomiting, and other hazards of polycystic kidney disease, renal insufficiency appear for.
Cystic kidney was the most common hazards? Understanding of the hazards of the polycystic kidney, polycystic kidney disease how to treat it? To penetrate therapy micro-based traditional Chinese medicine is the original Chinese medicine treatment by the hospital after years of research, disease outside the governance. It will be micro-processing of Chinese medicine through the skin penetration of the way so that the wall of blood vessels to dilate, to accelerate local blood circulation, quickly taken away by the cystic fluid at the same time, reduce the intracapsular pressure, inhibition of cyst epithelial cells continue to secrete, to reduce the cyst fluid generated, so that the cyst continuous retraction, can be effective in preventing cyst increases, reducing the damage of intrinsic renal cells.